Two cases presenting the syndrome of glaucomatocyclitic crises are reported. The history of one case extends over a period of 32 years. Mild aqueous flares were present in one case, but at no stage during the course of attacks in either case were keratic precipitates present. Surgery does not prevent recurrence of attacks. Topical treatment does not seem to shorten the course of a crisis. An hypothesis is advanced to account for the pathological and clinical features of this syndrome of unknown etiology and benign prognosis. The syndrome was recognized, although not published, in 1920, under the label of recurrent unilateral ocular hypertension of a cyclitic origin.
Attention is drawn to the phenomenon of pain referred to the ear from distant lesions. The various nervous arcs through which the reflex may be transmitted are indicated briefly. The great value of this reflex as an index of hidden disease is not sufficiently appreciated and a plea is made for a thorough ear, nose and throat examination in any case of persistent unilateral otalgia. Referred pain in the ear may be a rare presenting symptom of carcinoma of the lung. Stimuli may pass with facility in either direction through that particular reflex arc consisting of branches of the vagus so that while lesions in the areas supplied by the vagus may give rise to pain in the ear, conversely, any fret of the external auditory canal may initiate a cough, nausea, or even changes in cardiac rhythm.
In order to study changes in arterial oxygen saturation during anaesthesia without recourse to direct arterial sampling, we have constructed an oximeter similar to that descriptionbed by Wood and Geraci. Construction (rather than importation) was undertaken for reasons of economy. The principles of light transmission and measurement involved in oximetry are discussed briefly and the design of our oximeter is descriptionbed.